forum Paste the Last Thing that You Copied
Started by @CW-BornConfuzzledLeftILoveYa
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Snowflake you gotta stalker! ^~^
i
kinda
maybe
sorta
definitely
kinda
ship

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I'm just wondering if I should past the last thing I copied…. Its so long :(

JUST DO IT NI-NI

@TheGoldenLegend

Ok……
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Hemlock/Cicuta
Info about plant-
All members of Cicuta except C. bulbifera contain high levels of the poisonous principle cicutoxin, an unsaturated aliphatic alcohol that is structurally closely related to the toxin oenanthotoxin found in the plant hemlock water dropwort. Its primary toxic effect is to act as a stimulant in the central nervous system. It is a non-competitive gamma-aminobutyric acid (GABA) receptor antagonist.
The exact toxic dose of plant material in humans is unknown; it is thought ingestion of water hemlock in any quantity can result in poisoning and very small amounts may lead to death. Intoxication has also been reported following skin contact with the plant; a case was reported where a family of five people rubbed the plant onto the skin and were poisoned, with two children dying. Roots exposed by ploughing can also be the source of livestock poisonings Ingestion of plant material may cause death in the animal in as little as 15 minutes
Symptoms
The onset of symptoms following ingestion may be as soon as 15 minutes post ingestion. Nausea, Seizures, vomiting, abdominal pain, tremors, confusion, weakness, dizziness, and drowsiness;Complications of ongoing seizure activity include increased body temperature, decreases in the pH of the blood (metabolic acidosis), swelling in the brain, blood coagulation disorders, muscle breakdown (rhabdomyolysis), and kidney failure. Additional neurological symptoms may include hallucinations, delirium, tingling, pricking, or numbness of a person's skin, dilated pupils, and coma. Cardiovascular symptoms include alternating slow or fast heart rate and alternating low and high blood pressure. Other cardiac effects may include ECG (Electrocardiography) abnormalities such as widening of the PR interval, supraventricular tachycardia, and ventricular fibrillation. Symptoms of excess salivation, wheezing, respiratory distress, and absence of breathing have also been reported.
Deaths usually occur from respiratory failure or ventricular fibrillation secondary to ongoing seizure activity; fatalities have occurred within a few hours of ingestion. Poisoned people who recover usually regain consciousness and seizures cease within 24 to 48 hours of poisoning, although seizures may persist for up to 96 hours.There are occasional long-term effects such as retrograde amnesia of the events leading to intoxication and the intoxication itself. Other ongoing mild effects may include restlessness, muscle weakness, twitching, and anxiety. Complete resolution of symptoms may take a number of days or, in some cases, these ongoing symptoms may persist for months after poisoning.
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Oleander/Nerium oleander
Info about plant-
Oleander has historically been considered a poisonous plant because some of its compounds may exhibit toxicity, especially to animals, when consumed in large amounts. Among these compounds are oleandrin and oleandrigenin, known as cardiac glycosides, which are known to have a narrow therapeutic index and can be toxic when ingested.
Oleander is a common cause of poisoning and death in tropical and subtropical countries, with 170 cases seen at a single hospital in Sri Lanka. However, prognosis is good if the patient can reach a hospital in time; several common drugs can be used to stabilize the patient and counteract the glycosides. In reviewing oleander toxicity cases seen in-hospital, Lanford and Boor concluded that, except for children who might be at greater risk, "the human mortality associated with oleander ingestion is generally very low, even in cases of moderate intentional consumption (suicide attempts)".
Effects of poisoning
Ingestion of this plant can affect the gastrointestinal system, the heart, and the central nervous system. The gastrointestinal effects can consist of nausea and vomiting, excess salivation, abdominal pain, diarrhea that may contain blood, and especially in horses, colic. Cardiac reactions consist of irregular heart rate, sometimes characterized by a racing heart at first that then slows to below normal further along in the reaction. Extremities may become pale and cold due to poor or irregular circulation. The effect on the central nervous system may show itself in symptoms such as drowsiness, tremors or shaking of the muscles, seizures, collapse, and even coma that can lead to death.
Oleander sap can cause skin irritations, severe eye inflammation and irritation, and allergic reactions characterized by dermatitis.
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White snakeroot/Ageratina altissima
Toxicity
White snakeroot contains the toxin tremetol; when the plants are consumed by cattle, the meat and milk become contaminated with the toxin. When milk or meat containing the toxin is consumed, the poison is passed on to humans. If consumed in large enough quantities, it can cause tremetol poisoning in humans. The poisoning is also called milk sickness, as humans often ingested the toxin by drinking the milk of cows that had eaten snakeroot.
Symptoms in humans include loss of appetite, nausea, weakness, abdominal discomfort, reddened tongue and mucous membranes, and abnormal acidity of the blood, with accumulation of ketone bodies. In instances of serious poisoning, convulsions and coma may culminate in death. In persons who survive, weakness may persist for weeks.
The plants are also poisonous to horses, goats, and sheep. Signs of poisoning in these animals include depression and lethargy, placement of hind feet close together (horses, goats, cattle) or held far apart (sheep), nasal discharge, excessive salivation, arched body posture, and rapid or difficult breathing.
This plant may serve medicinal purposes. Root tea has been used to treat diarrhea, kidney stones, and fever. A root poultice can be used on snakebites.
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Monkshood/Aconitum
Toxicology
Marked symptoms may appear almost immediately, usually not later than one hour, and "with large doses death is almost instantaneous". Death usually occurs within two to six hours in fatal poisoning (20 to 40 ml of tincture may prove fatal). The initial signs are gastrointestinal, including nausea, vomiting, and diarrhea. This is followed by a sensation of burning, tingling, and numbness in the mouth and face, and of burning in the abdomen. In severe poisonings, pronounced motor weakness occurs and cutaneous sensations of tingling and numbness spread to the limbs. Cardiovascular features include hypotension, sinus bradycardia, and ventricular arrhythmias. Other features may include sweating, dizziness, difficulty in breathing, headache, and confusion. The main causes of death are ventricular arrhythmias and asystole, or paralysis of the heart or respiratory center.The only post mortem signs are those of asphyxia.
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Angel's Trumpet/Brugmansia
Toxicity
All parts of Brugmansia are potentially poisonous, with the seeds and leaves being especially dangerous. Brugmansia are rich in scopolamine (hyoscine), hyoscyamine, and several other tropane alkaloids.
Effects of ingestion can include paralysis of smooth muscles, confusion, tachycardia, dry mouth, diarrhea, migraine headaches, visual and auditory hallucinations, mydriasis, rapid onset cycloplegia, and death
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Rosary Pea/Abrus precatorius
Info about plant-
Abrus precatorius, known commonly as jequirity, Crab's eye, or crab's eye creeper, cock's eyes, rosary pea, paternoster pea, love pea, ext, is a slender, perennial climber that twines around trees, shrubs, and hedges. It is a legume with long, pinnate-leafleted leaves.
The plant is best known for its seeds, which are used as beads and in percussion instruments, and which are toxic because of the presence of abrin. Ingestion of a single seed, well chewed, can be fatal to both adults and children. The plant is native to a number of tropical areas. It has a tendency to become weedy and invasive where it has been introduced.
This plant is also poisonous to horses.
Symptoms of poisoning include
nausea
vomiting
convulsions
liver failure
death, usually after several days.
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Manchineel
Info about plant-
The manchineel tree (Hippomane mancinella) is a species of flowering plant in the spurge family (Euphorbiaceae). It is native to tropical southern North America and northern South America.
The name "manchineel" (sometimes written "manchioneel") as well as the specific epithet mancinella is from Spanish manzanilla ("little apple"), from the superficial resemblance of its fruit and leaves to those of an apple tree. A present-day Spanish name is in fact manzanilla de la muerte, "little apple of death". This refers to the fact that manchineel is one of the most dangerous trees in the world. Manchineel is also known as the beach apple.
Toxicity
All parts of the tree contain strong toxins, some unidentified.Its milky white sap contains phorbol and other skin irritants, producing strong allergic contact dermatitis. Standing beneath the tree during rain will cause blistering of the skin from mere contact with this liquid (even a small drop of rain with the milky substance in it will cause the skin to blister). The sap has also been known to damage the paint on cars. Burning the tree may cause ocular injuries if the smoke reaches the eyes. Contact with its milky sap (latex) produces bullous dermatitis, acute keratoconjunctivitis and possibly large corneal epithelial defects.
The fruit is possibly fatal if eaten; however, "fatalities from ingestion are not reported in the modern literature" and "ingestion may produce severe gastroenteritis with bleeding, shock, bacterial superinfection, and the potential for airway compromise due to edema. Patients with a history of ingestion and either oropharyngeal burns or gastrointestinal symptoms should be evaluated for admission into hospital. Care is supportive."
When ingested, the fruit is reportedly "pleasantly sweet" at first, with a subsequent "strange peppery feeling …, gradually progress[ing] to a burning, tearing sensation and tightness of the throat". Symptoms continue to worsen until the patient can "barely swallow solid food because of the excruciating pain and the feeling of a huge obstructing pharyngeal lump".
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The Suicide Tree/Cerbera odollam
Toxicity
The kernels of C. odollam contain cerberin, a digoxin-type cardenolide and cardiac glycoside toxin that blocks the calcium ion channels in heart muscle, causing disruption of the heart beat, most often fatally. Around half of the patients develops thrombocytopenia. Temporary cardiac pacing has been used in the management, apart from other supportive measures. The difficulty in detecting cerberin in autopsies and the ability of strong spices to mask its taste makes it an agent of homicide and suicide in India; there were more than 500 cases of fatal Cerbera poisoning between 1989 and 1999 in the southwest Indian state of Kerala.
A fatal dose of the poison is contained in one kernel, leading to death within 1–2 days.
Common symptoms include:
burning sensation in mouth
expurgation
violent vomiting
irregular respiration
headache
irregular heartbeat
coma and eventual death
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Deadly Nightshade/Atropa belladonna
Info about plant-
Atropa belladonna, commonly known as belladonna or deadly nightshade, is a perennial herbaceous plant in the nightshade family Solanaceae, which includes tomatoes, potatoes, and aubergine. It is native to Europe, North Africa, and Western Asia. Its distribution extends from Great Britain in the west to western Ukraine and the Iranian province of Gilan in the east. It is also naturalised and/or introduced in some parts of Canada and the United States.
The foliage and berries are extremely toxic when ingested, containing tropane alkaloids. These toxins include atropine, scopolamine and hyoscyamine, which cause delirium and hallucinations, and are also used as pharmaceutical anticholinergics.
The antidote for belladonna poisoning is physostigmine or pilocarpine, the same as for atropine
Toxicity
Belladonna is one of the most toxic plants found in the Eastern Hemisphere,and its use by mouth increases risk in numerous clinical conditions, such as complications of pregnancy, cardiovascular diseases, gastrointestinal disorders, and psychiatric disorders, among others. All parts of the plant contain tropane alkaloids. The berries pose the greatest danger to children because they look attractive and have a somewhat sweet taste. The root of the plant is generally the most toxic part, though this can vary from one specimen to another.
The symptoms of belladonna poisoning include dilated pupils, sensitivity to light, blurred vision, tachycardia, loss of balance, staggering, headache, rash, flushing, severely dry mouth and throat, slurred speech, urinary retention, constipation, confusion, hallucinations, delirium, and convulsions. In 2009, A. belladonna berries were mistaken for blueberries by an adult woman; the six berries she ate were documented to result in severe anticholinergic syndrome. The plant's deadly symptoms are caused by atropine's disruption of the parasympathetic nervous system's ability to regulate involuntary activities, such as sweating, breathing, and heart rate. The antidote for belladonna poisoning is physostigmine or pilocarpine, the same as for atropine.
Atropa belladonna is also toxic to many domestic animals, causing narcosis and paralysis.However, cattle and rabbits eat the plant seemingly without suffering harmful effects. In humans, its anticholinergic properties will cause the disruption of cognitive capacities, such as memory and learning.
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Purple cockle/Agrostemmagithago
Info about plant-
Agrostemma githago, the common corn-cockle (also written "corncockle" and "corn cockle" and known locally simply as "the corncockle"). The species is a weed of cereals and other crops, probably with a centre of origin in the eastern Mediterranean. Nowadays declining in its native range because of improved seed cleaning, it is found as a weed worldwide. Agrostemma gracile, the slender corncockle, is only found in central Greece near the city of Farsala. Corncockle is an attractive plant, and its seeds are still commercially available to gardeners. All parts of plant are poisonous if ingested.
Toxicity
All parts of the plant are poisonous (githagin, agrostemmic acid). It has been used in folk medicine despite the risk of fatal poisoning.
· abundant salivation
· sore throat
· aches
· nausea
· cramping
· headache
· dizziness
· fever
· agitation
· Abdominal pain
· Vomiting
· Diarrhea
· Slowed breathing
· Weakness
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Jimsonweed/Datura stramonium
Toxicity
All parts of Datura plants contain dangerous levels of the tropane alkaloids atropine, hyoscyamine, and scopolamine, which are classified as deliriants, or anticholinergics. The risk of fatal overdose is high among uninformed users, and many hospitalizations occur amongst recreational users who ingest the plant for its psychoactive effects.
The amount of toxins varies widely from plant to plant.
Datura intoxication typically produces delirium (as contrasted to hallucination), hyperthermia, tachycardia, bizarre behavior, and severe mydriasis with resultant painful photophobia that can last several days. Pronounced amnesia is another commonly reported effect. The onset of symptoms generally occurs around 30 to 60 minutes after ingesting the herb. These symptoms generally last from 24 to 48 hours, but have been reported in some cases to last as long as two weeks.
Better list of Symptoms-
Hot skin
Dry skin
Flushed skin
Dilated pupils
Headache
Coma
Reduced bowel sounds
Disorientation
Dizziness
High blood pressure
Reduced gastrointestinal motility
Nausea
Vomiting
Seizures
Rapid heart rate
Urinary retention
Visual hallucinations
As with other cases of anticholinergic poisoning, intravenous physostigmine can be administered in severe cases as an antidote.
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Daphne (plant)
Daphne (/ˈdæfniː/; Greek: Δάφνη, meaning "laurel") is a genus of between 70 and 95 species of deciduous and evergreen shrubs in the family Thymelaeaceae, native to Asia, Europe and north Africa. They are noted for their scented flowers and often brightly coloured berries. Two species are used to make paper. Many species are grown in gardens as ornamental plants; the smaller species are often used in rock gardens. All parts of daphnes are poisonous, especially the berries
Toxicity
All parts of daphnes are toxic, the berries being particularly so. One active compound is daphnin, a glycoside, combining glucose with daphnetin. Some species have been shown to contain a further toxin, mezerein.
Symptoms of ingestion include-
Lesions of the mouth
and in severe cases damage to the kidneys (nephritis),
Irregular heart rhythm
Inflammation
Skin irritation
Itchy skin
Eye irritation
Localized skin swelling, redness, and blistering
Nausea
Vomiting
Gastroenteritis
Temporary blindness
Burning mouth and throat
Headache
Delirium
Coma.

Deleted user

Divergent by Veronica Roth (487) 3 stars
Summer of Salt Katrina Leno (272) 3 stars
Flight Risk by Jennifer Fenn (256) 2 stars
The first four books in the Keeper Of The Lost Cities series by Sarah Messenger ( around 3,000 pages maybe?) 3 stars
The Body Market by Donna Freitas (409)
The Lost Causes by Jessica Etting (341 pages) 5 Stars!
The Upside of unrequited by Becky Albertalli (336) 5 stars!
No Summit out of Sight by Jordan Romero (383) 5 stars!!!
Ship It by Britta Lundin (384) 6 STARS!!!!!

@Sugar-Lover

A grin appears on his face. "I love you."

Yoongi almost did some sort of crazy reaction but knew it would only make Jimin's condition worse. "Then let's get you some food."

"What could I possibly get in trouble for? I've been good lately."

@I-like-writing-horror-stories-so-fear-me

Harry Potter and the portrait of what looked like a large pile of ash

Chapter 13: The Handsome One

The castle grounds snarled with a wave of magically magnified wind. The sky outside was a great black ceiling, which was full of blood. The only sounds drifting from Hagrid’s hut were the disdainful shrieks of his own furniture. Magic: it was something that Harry Potter thought was very good.
Leathery sheets of rain lashed at Harry’s ghost as he walked across the grounds toward the castle. Ron was standing there and doing a kind of frenzied tap dance. He saw Harry and immediately began to eat Hermione's family.

Ron’s Ron shirt was just as bad as Ron himself.

“If you two can’t clump happily, I’m going to get aggressive,” confessed the reasonable Hermione.
“What about Ron magic?” offered Ron. To Harry, Ron was a loud, slow, and soft bird. Harry did not like to think about birds.
“Death Eaters are on top of the castle!” Ron bleated, quivering. Ron was going to be spiders. He just was. He wasn’t proud of that, but it was going to be hard to not have spiders all over his body when all is said and done.
“Look,” said Hermione. “Obviously there are loads of Death Eaters in the castle. Let’s listen in on their meetings.”
The three complete friends zapped onto the landing outside the door to the castle roof. They almost legged it, but witches are not climbing. Ron looked at the doorknob and then looked at Hermione with searing pain.
“I think it’s closed,” he noticed. “Locked,” said Mr. Staircase, the shabby-robed ghost. They looked at the door, screaming about how closed it was and asking it to be replaced with a small orb. The password was “BEEF WOMEN,” Hermione cried.
Harry, Ron, and Hermione quietly stood behind a circle of Death Eaters who looked bad.
“I think it’s okay if you like me,” said one Death Eater.
“Thank you very much,” replied the other. The first Death Eater confidently leaned forward to plant a kiss on his cheek.
“Oh! Well done!” said the second as his friend stepped back again. All the other Death Eaters clapped politely. Then they all took a few minutes to go over the plan to get rid of Harry Potter’s magic.
Harry could tell that Voldemort was standing right behind him. He felt a great overreaction. Harry tore his eyes from his head and threw them in the forest. Voldemort raised his eyebrows at Harry, who could not see anything at the moment.
“Voldemort, you’re a very bad and mean wizard,” Harry savagely said. Hermione nodded encouragingly. The tall Death Eater was wearing a shirt that said “Hermione Has Forgotten How To Dance,” so Hermione dipped his head in mud.
Ron threw a wand at Voldemort and everyone applauded. Ron smiled. Ron reached for his wand slowly.
“Ron’s the handsome one,” muttered Harry as he reluctantly reached for his. They cast a spell or two, and jets of green light shot out of the Death Eater’s heads. Ron flinched.
“Not so handsome now,” thought Harry as he dipped Hermione in hot sauce. The Death Eaters were dead now, and Harry was hungrier than he had ever been.
The pig of Hufflepuff pulsed like a large bullfrog. Dumbledore smiled at it, and placed his hand on its head: “You are Hagrid now.”
“We’re the only people who matter. He’s not going to get rid of us,” Harry, Hermione, and Ron sang in chorus.
The floor of the castle seemed like a large pile of magic. The Dursleys had never been to the castle and they were not about to come there in Harry Potter and the Portrait of What Looked Like a Large Pile of Ash. Harry looked around and then fell down the spiral staircase for the rest of the summer.
“I’m Harry Potter,” Harry began yelling. “The dark arts better be worried, oh boy!”

The End

@Imperfect_Autumn group

Kasey saw the look in her eyes and heard her stutter. Her expression softened, and she held a hand out to her to hold her hand.

"I'm sorry," she said softly. "But something did come out of it, whether that's good or not is up to you. You have a superpower, Kitty."